Neurological Rehabilitation
Conditions We Treat
Browse the conditions we specialize in
Drop Foot Syndrome
What Is Drop Foot Syndrome? Drop foot syndrome is a neurological condition caused by damage to the peroneal nerve that makes it difficult or impossible for a person to lift the front part of the foot. This leads to a gait disturbance; the patient drags the foot while walking or raises the leg abnormally high to avoid stumbling (steppage gait). It is less a disease in itself and more a sign of an underlying neurological or nerve problem. What Are the Causes? Any condition that compresses or damages the peroneal nerve can cause drop foot: Stroke (brain or spinal cord origin) Multiple Sclerosis (MS) Lumbar disc herniation with nerve root compression Trauma or surgery around the knee Habitual leg-crossing Diabetic peripheral neuropathy Charcot-Marie-Tooth disease Treatment Approach at ROMMER At ROMMER, each patient's clinical picture, underlying cause, and quality-of-life goals are evaluated together to create a personalised rehabilitation programme: Functional Electrical Stimulation (FES): Low-frequency electrical current stimulates the peroneal nerve to retrain the foot-lift movement. Ankle-Foot Orthosis (AFO): An appropriate orthosis is selected to ensure safe gait and reduce fall risk. Neuromuscular Re-education: Proprioception and coordination exercises strengthen the nerve-muscle connection. Balance and Gait Training: Patient education and assistive device adaptation are provided to establish a safe walking pattern. Botulinum Toxin (Botox): Applied when necessary to reduce antagonist muscle spasm and support functional gains. What to Expect During Recovery? Rehabilitation started early can accelerate peroneal nerve regeneration and increase functional gains. Depending on the degree of nerve damage and the underlying cause, full recovery is possible in some patients, while in others significant progress is made in maintaining gait safety and quality of life.
Facial Palsy
What Is Facial Palsy (Facial Paralysis)? Facial palsy is a condition caused by damage to the 7th cranial nerve (facial nerve), which controls the muscles of facial expression, resulting in loss of movement on one or both sides of the face. Inability to close the eyelid, drooping of the corner of the mouth, difficulty retaining food in the mouth, and speech difficulties are among the most common symptoms. What Are the Causes? Bell's Palsy: The most common type; usually develops due to inflammation of the facial nerve following a viral infection. Stroke: Central facial palsy can occur in strokes of central nervous system origin. Ramsay Hunt Syndrome: Facial paresis due to herpes zoster infection affecting the ear region. Trauma: Nerve injury during skull base fractures or surgery. Tumour: Masses such as parotid gland tumours or acoustic neuromas compressing the facial nerve. Facial Palsy Rehabilitation at ROMMER The goal of rehabilitation in facial palsy is not only cosmetic; it is to regain critical functions such as swallowing, speech, and eye protection. Key components of the programme applied at ROMMER: Neuromuscular Electrical Stimulation (NMES): Fine electrical impulses delivered to the paralysed facial muscles re-stimulate the nerve-muscle connection. Mirror Therapy: Movements of the healthy side are reflected back to the brain as "healthy" input through a mirror, supporting motor learning. Facial Expression Exercises: Specific movements such as raising the eyebrow, blinking, pursing the lips, and smiling are practised progressively. Manual Therapy and Massage: Applied to maintain tone balance of the facial muscles and prevent the development of synkinesis. Swallowing Therapy: In cases of dysphagia, a specialised programme is created in collaboration with a speech-language therapist. Botulinum Toxin: Can be applied selectively in cases of synkinesis or abnormal increase in muscle tone. Recovery Process In idiopathic forms such as Bell's palsy, the vast majority of patients show significant improvement within the first 3–6 months. The process may be longer in strokes or trauma-related facial palsy, and regular rehabilitation is critical. At ROMMER, the programme is updated weekly according to each patient's rate of recovery; our goal is to return to social life and confidence as quickly as possible.
Spinal Cord Injuries
What Are Spinal Cord Injuries? Spinal cord injuries occur as a result of damage to the nerve tissue within the spine due to trauma, disease, or vascular causes. Depending on the level of injury and whether it is complete (full disruption) or incomplete (partial), patients may experience varying degrees of loss of movement, loss of sensation, and involvement of the autonomic nervous system. Neck-level injuries can result in tetraplegia (involvement of 4 limbs), while lumbar-level injuries can result in paraplegia (involvement of both legs). Causes of Spinal Cord Injuries Traffic accidents Falls from height Sports injuries (diving, windsurfing, etc.) Gunshot wounds and penetrating/cutting trauma Tumours and spinal metastases Spinal infections (spondylodiscitis) Vascular events (spinal infarction) Spinal Cord Injury Rehabilitation at ROMMER Following a spinal cord injury, rehabilitation is planned to maximise the patient's remaining neurological potential, prevent complications, and develop independent living skills. ROMMER's multidisciplinary team manages the following components together: Physical Strengthening Programme: An intensive exercise programme is implemented to strengthen preserved muscle groups and develop upper limb independence. Functional Electrical Stimulation (FES): Electrical stimulation to dysfunctional muscles supports both strength and cardiovascular conditioning. Robotic Gait Training: Intensive gait rehabilitation stimulating neural plasticity is applied with robotic devices. Occupational Therapy and Daily Living Training: Wheelchair use, transfer techniques, bathroom/toilet independence, and home adaptation are provided. Respiratory Rehabilitation: Strengthening of respiratory muscles and secretion management are planned especially for high cervical injuries. Neurogenic Bladder-Bowel Management: In collaboration with urology and gastroenterology, an intermittent catheterisation, nutrition, and bowel training programme is implemented. Psychosocial Support: Psychiatric and clinical psychology support is provided to help patients and their families cope with the psychological burden of injury. Neurological Recovery and Expectations The majority of neurological recovery occurs within the first 6–12 months; therefore, intensive early-phase rehabilitation is the most critical period. In incomplete injuries, the potential for functional gain is higher. In complete injuries, the goal is to develop independent living skills, prevent complications, and maintain quality of life. At ROMMER, realistic and achievable goals are set for each patient, progress is measured regularly, and the programme is updated accordingly.
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